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Nitric oxide promotes GABA release by activating a voltage-independent Ca(2+) influx pathway in retinal amacrine cells
Retinal amacrine cells express nitric oxide (NO) synthase and produce NO, making NO available to regulate the function of amacrine cells. Here we test the hypothesis that NO can alter the GABAergic synaptic output of amacrine cells. We investigate this using whole cell voltage clamp recordings and C...
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| Publicado en: | J Neurophysiol |
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| Autores principales: | , |
| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
American Physiological Society
2017
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5340885/ https://ncbi.nlm.nih.gov/pubmed/28053242 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/jn.00803.2016 |
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