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Deletion of protein kinase C-ε attenuates mitochondrial dysfunction and ameliorates ischemic renal injury
Previously, we documented that activation of protein kinase C-ε (PKC-ε) mediates mitochondrial dysfunction in cultured renal proximal tubule cells (RPTC). This study tested whether deletion of PKC-ε decreases dysfunction of renal cortical mitochondria and improves kidney function after renal ischemi...
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| Foilsithe in: | Am J Physiol Renal Physiol |
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| Main Authors: | , , |
| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
American Physiological Society
2016
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5283890/ https://ncbi.nlm.nih.gov/pubmed/27760765 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00115.2016 |
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