FUS3 represses CLN1 and CLN2 and in concert with KSS1 promotes signal transduction.
FUS3 is functionally redundant with KSS1, a homologous yeast protein kinase, for a step(s) in signal transduction between the beta subunit of the guanine nucleotide binding protein (G protein), STE4, and the mating type-specific transcriptional activator, STE12. Either FUS3 or KSS1 can execute this...
שמור ב:
| הוצא לאור ב: | Proc Natl Acad Sci U S A |
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| Principais autores: | , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
National Academy of Sciences
1991
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC52723/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1946350/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.88.21.9392 |
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