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Variation in stiffness regulates cardiac myocyte hypertrophy via signaling pathways
Much diseased human myocardial tissue is fibrotic and stiff, which increases the work that the ventricular myocytes must perform to maintain cardiac output. The hypothesis tested is that the increased load due to greater stiffness of the substrata drives sarcomere assembly of cells, thus strengtheni...
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| Publicado no: | Can J Physiol Pharmacol |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5222882/ https://ncbi.nlm.nih.gov/pubmed/27486838 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1139/cjpp-2015-0578 |
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