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Tat-HSP22 inhibits oxidative stress-induced hippocampal neuronal cell death by regulation of the mitochondrial pathway

Oxidative stress plays an important role in the progression of various neuronal diseases including ischemia. Heat shock protein 22 (HSP22) is known to protect cells against oxidative stress. However, the protective effects and mechanisms of HSP22 in hippocampal neuronal cells under oxidative stress...

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التفاصيل البيبلوغرافية
الحاوية / القاعدة:	Mol Brain
المؤلفون الرئيسيون:	Jo, Hyo Sang, Kim, Dae Won, Shin, Min Jea, Cho, Su Bin, Park, Jung Hwan, Lee, Chi Hern, Yeo, Eun Ji, Choi, Yeon Joo, Yeo, Hyeon Ji, Sohn, Eun Jeong, Son, Ora, Cho, Sung-Woo, Kim, Duk-Soo, Yu, Yeon Hee, Lee, Keun Wook, Park, Jinseu, Eum, Won Sik, Choi, Soo Young
التنسيق:	Artigo
اللغة:	Inglês
منشور في:	BioMed Central 2017
الموضوعات:	Research
الوصول للمادة أونلاين:	https://ncbi.nlm.nih.gov/pmc/articles/PMC5210279/ https://ncbi.nlm.nih.gov/pubmed/28052764 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13041-016-0281-8
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Tat-HSP22 inhibits oxidative stress-induced hippocampal neuronal cell death by regulation of the mitochondrial pathway

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