Functional complementation of ataxia-telangiectasia group D (AT-D) cells by microcell-mediated chromosome transfer and mapping of the AT-D locus to the region 11q22-23.

The hereditary human disease ataxia-telangiectasia (AT) is characterized by phenotypic complexity at the cellular level. We show that multiple mutant phenotypes of immortalized AT cells from genetic complementation group D (AT-D) are corrected after the introduction of a single human chromosome from...

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Hlavní autoři: Lambert, C, Schultz, R A, Smith, M, Wagner-McPherson, C, McDaniel, L D, Donlon, T, Stanbridge, E J, Friedberg, E C
Médium: Artigo
Jazyk:Inglês
Vydáno: 1991
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Research Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC51987/
https://ncbi.nlm.nih.gov/pubmed/2062869
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