Wild-type and e antigen-minus hepatitis B viruses and course of chronic hepatitis.
Using an oligonucleotide hybridization assay, we studied the clinical implication of wild-type hepatitis B virus (HBV) and a HBV mutant that is unable to secrete hepatitis B e antigen (HBeAg) because of a translational defect due to a stop codon in the pre-C region in 106 hepatitis B surface antigen...
Uloženo v:
| Vydáno v: | Proc Natl Acad Sci U S A |
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| Hlavní autoři: | , , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
National Academy of Sciences
1991
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC51623/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2034663/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.88.10.4186 |
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