Glucose metabolism reprogrammed by overexpression of IKK-epsilon promotes pancreatic tumor growth

Aberrant expression of the kinase IKKε in pancreatic ductal adenocarcinoma (PDAC) has been associated with poor prognosis. In this study, we define a pathobiologic function for IKKε in reprogramming glucose metabolism and driving progression in PDAC. Silencing IKKε in PDAC cells, which overexpressed...

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Publicat a:Cancer Res
Autors principals: Zubair, Haseeb, Azim, Shafquat, Srivastava, Sanjeev Kumar, Ahmad, Aamir, Bhardwaj, Arun, Khan, Mohammad Aslam, Patel, Girijesh Kumar, Arora, Sumit, Carter, James Elliot, Singh, Seema, Singh, Ajay Pratap
Format: Artigo
Idioma:Inglês
Publicat: 2016
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Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC5161695/
https://ncbi.nlm.nih.gov/pubmed/27923829
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/0008-5472.CAN-16-1666
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