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Ranolazine for Congenital Long QT Syndrome Type III (LQT3): Experimental and Long-Term Clinical Data

BACKGROUND: The basic defect in long QT syndrome type-III (LQT3) is an excessive inflow of sodium current during phase-3 of the action potential caused by mutations in the SCN5A gene. Most sodium channel blockers reduce the early (peak) and late components of the sodium current (I(Na) and I(NaL)) bu...

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Bibliografske podrobnosti
izdano v:	Circ Arrhythm Electrophysiol
Main Authors:	Chorin, Ehud, Hu, Dan, Antzelevitch, Charles, Hochstadt, Aviram, Belardinelli, Luiz, Zeltser, David, Barajas-Martinez, Hector, Rozovski, Uri, Rosso, Raphael, Adler, Arnon, Benhorin, Jesaia, Viskin, Sami
Format:	Artigo
Jezik:	Inglês
Izdano:	2016
Teme:	Article
Online dostop:	https://ncbi.nlm.nih.gov/pmc/articles/PMC5119553/ https://ncbi.nlm.nih.gov/pubmed/27733495 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCEP.116.004370
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Ranolazine for Congenital Long QT Syndrome Type III (LQT3): Experimental and Long-Term Clinical Data

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