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Ranolazine for Congenital Long QT Syndrome Type III (LQT3): Experimental and Long-Term Clinical Data
BACKGROUND: The basic defect in long QT syndrome type-III (LQT3) is an excessive inflow of sodium current during phase-3 of the action potential caused by mutations in the SCN5A gene. Most sodium channel blockers reduce the early (peak) and late components of the sodium current (I(Na) and I(NaL)) bu...
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| 出版年: | Circ Arrhythm Electrophysiol |
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| 主要な著者: | , , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5119553/ https://ncbi.nlm.nih.gov/pubmed/27733495 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCEP.116.004370 |
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