Metformin Ameliorates Dysfunctional Traits of Glibenclamide- and Glucose-Induced Insulin Secretion by Suppression of Imposed Overactivity of the Islet Nitric Oxide Synthase-NO System

Metformin lowers diabetic blood glucose primarily by reducing hepatic gluconeogenesis and increasing peripheral glucose uptake. However, possible effects by metformin on beta-cell function are incompletely understood. We speculated that metformin might positively influence insulin secretion through...

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Опубликовано в: :PLoS One
Главные авторы: Lundquist, Ingmar, Mohammed Al-Amily, Israa, Meidute Abaraviciene, Sandra, Salehi, Albert
Формат: Artigo
Язык:Inglês
Опубликовано: Public Library of Science 2016
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Research Article
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC5098820/
https://ncbi.nlm.nih.gov/pubmed/27820841
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0165668
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