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Novel Toll-like receptor-4 antagonist (+)-naloxone protects mice from inflammation-induced preterm birth
Toll-like receptor 4 (TLR4) activation by bacterial infection, or by sterile inflammatory insult is a primary trigger of spontaneous preterm birth. Here we utilize mouse models to investigate the efficacy of a novel small molecule TLR4 antagonist, (+)-naloxone, the non-opioid isomer of the opioid re...
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| Publicado no: | Sci Rep |
|---|---|
| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5098167/ https://ncbi.nlm.nih.gov/pubmed/27819333 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/srep36112 |
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