Treatment with interferon-alpha preferentially reduces the capacity for amplification of granulocyte-macrophage progenitors (CFU-GM) from patients with chronic myeloid leukemia but spares normal CFU-GM.

The biological target for interferon (IFN)-alpha in chronic myeloid leukemia (CML) is unknown, but one possibility is that amplification of granulocyte-macrophage colony-forming cells (CFU-GM) is reduced. Replating CFU-GM colonies and observing secondary colony formation provides a measure of CFU-GM...

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Bibliografiska uppgifter
Huvudupphovsmän: Gordon, M Y, Marley, S B, Lewis, J L, Davidson, R J, Nguyen, D X, Grand, F H, Amos, T A, Goldman, J M
Materialtyp: Artigo
Språk:Inglês
Publicerad: 1998
Ämnen:
Research Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC508933/
https://ncbi.nlm.nih.gov/pubmed/9710439
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