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Reversal of impaired wound repair in iNOS-deficient mice by topical adenoviral-mediated iNOS gene transfer.

Most evidence indicates that nitric oxide plays a role in normal wound repair; however, involvement of inducible nitric oxide synthase (iNOS) has not been established. Experiments were carried out to determine the requirement for iNOS in closing excisional wounds. Wound closure was delayed by 31% in...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Yamasaki, K, Edington, H D, McClosky, C, Tzeng, E, Lizonova, A, Kovesdi, I, Steed, D L, Billiar, T R
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 1998
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC508647/
https://ncbi.nlm.nih.gov/pubmed/9486966
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