Differentiation of glucose toxicity from beta cell exhaustion during the evolution of defective insulin gene expression in the pancreatic islet cell line, HIT-T15.

Lignende værker

• Prevention of glucose toxicity in HIT-T15 cells and Zucker diabetic fatty rats by antioxidants
  af: Tanaka, Yoshito, et al.
  Udgivet: (1999)
• Rising Intracellular Zinc by Membrane Depolarization and Glucose in Insulin-Secreting Clonal HIT-T15 Beta Cells
  af: Slepchenko, Kira G., et al.
  Udgivet: (2012)
• Chronic exposure of betaTC-6 cells to supraphysiologic concentrations of glucose decreases binding of the RIPE3b1 insulin gene transcription activator.
  af: Poitout, V, et al.
  Udgivet: (1996)
• Somatostatin coordinately regulates glucagon gene expression and exocytosis in HIT-T15 cells.
  af: Kendall, D M, et al.
  Udgivet: (1995)
• Silymarin Activates c-AMP Phosphodiesterase and Stimulates Insulin Secretion in a Glucose-Dependent Manner in HIT-T15 Cells
  af: Meng, Ran, et al.
  Udgivet: (2016)