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Plasminogen activator inhibitor-1 in acute hyperoxic mouse lung injury.

Hyperoxia-induced lung disease is associated with prominent intraalveolar fibrin deposition. Fibrin turnover is tightly regulated by the concerted action of proteases and antiproteases, and inhibition of plasmin-mediated proteolysis could account for fibrin accumulation in lung alveoli. We show here...

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Detalles Bibliográficos
Publicado en:J Clin Invest
Principais autores: Barazzone, C, Belin, D, Piguet, P F, Vassalli, J D, Sappino, A P
Formato: Artigo
Idioma:Inglês
Publicado: American Society for Clinical Investigation 1996
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Acceso en liña:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC507728/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8981909/
https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI119089
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