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TLR2 activation limits rhinovirus-stimulated CXCL-10 by attenuating IRAK-1-dependent IL-33 receptor signaling in human bronchial epithelial cells
Airway epithelial cells are the major target for rhinovirus (RV) infection and express pro-inflammatory chemokines and antiviral cytokines that play a role in innate immunity. Previously, we demonstrated that RV interaction with TLR2 causes IRAK-1 depletion in both airway epithelial cells and macrop...
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| Publicado no: | J Immunol |
|---|---|
| Main Authors: | , , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5070654/ https://ncbi.nlm.nih.gov/pubmed/27503209 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1502702 |
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