The NK1R-/- mouse phenotype suggests that small body size, with a sex- and diet-dependent excess in body mass and fat, are physical biomarkers for a human endophenotype with vulnerability to attention deficit hyperactivity disorder

Lignende værker

• The angiotensin converting enzyme inhibitor, captopril, prevents the hyperactivity and impulsivity of neurokinin-1 receptor gene ‘knockout’ mice: Sex differences and implications for the treatment of attention deficit hyperactivity disorder
  af: Porter, Ashley J., et al.
  Udgivet: (2015)
• Behavioral neuroscience of attention deficit hyperactivity disorder and its treatment
  af: Stanford, Clare., et al.
  Udgivet: (2012.)
• Who First Suggests the Diagnosis of Attention-Deficit/Hyperactivity Disorder?
  af: Sax, Leonard, et al.
  Udgivet: (2003)
• Atomoxetine reduces hyperactive/impulsive behaviours in neurokinin-1 receptor ‘knockout’ mice
  af: Pillidge, Katharine, et al.
  Udgivet: (2014)
• Steroid sulfatase-deficient mice exhibit endophenotypes relevant to Attention Deficit Hyperactivity Disorder
  af: Trent, Simon, et al.
  Udgivet: (2012)