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Specific inhibition of c-Jun N-terminal kinase delays preterm labour and reduces mortality
Preterm labour (PTL) is commonly associated with infection and/or inflammation. Lipopolysaccharide (LPS) from different bacteria can be used to independently or mutually activate Jun N-terminal kinase (JNK)/AP1- or NF-κB-driven inflammatory pathways that lead to PTL. Previous studies using Salmonell...
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| 出版年: | Reproduction |
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| 主要な著者: | , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Bioscientifica Ltd
2015
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4982111/ https://ncbi.nlm.nih.gov/pubmed/26183892 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1530/REP-15-0258 |
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