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Suppression of Resting Metabolism by the Angiotensin AT(2) Receptor

Activation of the brain renin-angiotensin system (RAS) stimulates energy expenditure through increasing resting metabolic rate (RMR), and this effect requires simultaneous suppression of the circulating/adipose RAS. To identify the mechanism by which the peripheral RAS opposes RMR control by the bra...

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Dettagli Bibliografici
Pubblicato in:Cell Rep
Autori principali: Littlejohn, Nicole K., Keen, Henry L., Weidemann, Benjamin J., Claflin, Kristin E., Tobin, Kevin V., Markan, Kathleen R., Park, Sungmi, Naber, Meghan C., Gourronc, Francoise A., Pearson, Nicole A., Liu, Xuebo, Morgan, Donald A., Klingelhutz, Aloysius J., Potthoff, Matthew J., Rahmouni, Kamal, Sigmund, Curt D., Grobe, Justin L.
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2016
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4981564/
https://ncbi.nlm.nih.gov/pubmed/27477281
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.celrep.2016.07.003
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