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Nitric Oxide Stimulates Glutamatergic Synaptic Inputs to Baroreceptor Neurons Through Potentiation of Ca(v)2.2-mediated Ca(2+) Currents
Nitric oxide (NO) increases glutamate release to the second-order neurons in the nucleus tractus solitarius (NTS). N-type Ca(2+) channel is essential for triggering glutamate release at synaptic terminals. In this study, we determined the role of Ca(v)2.2 subunit in NO-induced increase in glutamate...
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| Опубликовано в: : | Neurosci Lett |
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| Главные авторы: | , |
| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
2014
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4978222/ https://ncbi.nlm.nih.gov/pubmed/24686191 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neulet.2014.03.036 |
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