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Activation of calpain by renin-angiotensin system in pleural mesothelial cells mediates tuberculous pleural fibrosis
Pleural fibrosis is defined as an excessive deposition of extracellular matrix (ECM) components that results in destruction of the normal pleural tissue architecture. It can result from diverse inflammatory conditions, especially tuberculous pleurisy. Pleural mesothelial cells (PMCs) play a pivotal...
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| Vydáno v: | Am J Physiol Lung Cell Mol Physiol |
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| Hlavní autoři: | , , , , , , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Physiological Society
2016
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4967195/ https://ncbi.nlm.nih.gov/pubmed/27261452 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00348.2015 |
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