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Impact of genotype on endocrinal complications in β-thalassemia patients
In β-thalassemia, certain mutations cause a complete absence of β-globin chain synthesis, termed β(0)-thalassemia, while others may allow certain β-globin production and are termed β(+)- or β(++)-thalassemia. The homozygous state results in severe anemia, which requires regular blood transfusion. By...
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| Publicat a: | Biomed Rep |
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| Autors principals: | , , , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
D.A. Spandidos
2016
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4887852/ https://ncbi.nlm.nih.gov/pubmed/27284414 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3892/br.2016.646 |
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