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Transcriptional Profiling of Rapamycin-Treated Fibroblasts From Hypertrophic and Keloid Scars
Excess scar formation after cutaneous injury can result in hypertrophic scar (HTS) or keloid formation. Modern strategies to treat pathologic scarring represent nontargeted approaches that produce suboptimal results. Mammalian target of rapamycin (mTOR), a central mediator of inflammation, has been...
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| Pubblicato in: | Ann Plast Surg |
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| Autori principali: | , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2014
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4886898/ https://ncbi.nlm.nih.gov/pubmed/24835866 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1097/SAP.0b013e31826956f6 |
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