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Control of Ca(2+) Influx and Calmodulin Activation by SK-Channels in Dendritic Spines
The key trigger for Hebbian synaptic plasticity is influx of Ca(2+) into postsynaptic dendritic spines. The magnitude of [Ca(2+)] increase caused by NMDA-receptor (NMDAR) and voltage-gated Ca(2+) -channel (VGCC) activation is thought to determine both the amplitude and direction of synaptic plastici...
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| 出版年: | PLoS Comput Biol |
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| 主要な著者: | , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Public Library of Science
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4883788/ https://ncbi.nlm.nih.gov/pubmed/27232631 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pcbi.1004949 |
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