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HDAC9 exacerbates endothelial injury in cerebral ischaemia/reperfusion injury
Histone deacetylase (HDAC) 9, a member of class II HDACs, regulates a wide variety of normal and abnormal physiological functions, which is usually expressed at high levels in the brain and skeletal muscle. Although studies have highlighted the importance of HDAC‐mediated epigenetic processes in the...
Uloženo v:
| Vydáno v: | J Cell Mol Med |
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| Hlavní autoři: | , , , , , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
John Wiley and Sons Inc.
2016
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4882992/ https://ncbi.nlm.nih.gov/pubmed/26865248 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.12803 |
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