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Deletion of LOX-1 Protects against Heart Failure Induced by Doxorubicin
Oxidative stress is one of the major factors in doxorubicin (DOX)-induced cardiomyopathy. Lectin-like oxidized low-density lipoprotein (oxLDL) receptor-1 (LOX-1) plays an important role to regulate cardiac remodeling and oxidative stress after ischemia-reperfusion. Therefore, we examined whether or...
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| Izdano u: | PLoS One |
|---|---|
| Glavni autori: | , , , , , , , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
Public Library of Science
2016
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4873018/ https://ncbi.nlm.nih.gov/pubmed/27195769 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0154994 |
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