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Laquinimod prevents cuprizone-induced demyelination independent of Toll-like receptor signaling
OBJECTIVE: To test whether Toll-like receptor (TLR) signaling plays a key role for reduced nuclear factor B (NF-κB) activation after laquinimod treatment in the model of cuprizone-induced demyelination, oligodendrocyte apoptosis, inflammation, and axonal damage. METHODS: Ten-week-old C57BL/6J, TLR4(...
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| Publicado no: | Neurol Neuroimmunol Neuroinflamm |
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| Main Authors: | , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Lippincott Williams & Wilkins
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4871804/ https://ncbi.nlm.nih.gov/pubmed/27231712 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1212/NXI.0000000000000233 |
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