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β-Arrestins promote podocyte injury by inhibition of autophagy in diabetic nephropathy
β-Arrestins are multifunctional proteins originally identified as negative adaptors of G protein-coupled receptors (GPCRs). Emerging evidence has also indicated that β-arrestins can activate signaling pathways independent of GPCR activation. This study was to elucidate the role of β-arrestins in dia...
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| 發表在: | Cell Death Dis |
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| Main Authors: | , , , , , , , , , , |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
Nature Publishing Group
2016
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4855668/ https://ncbi.nlm.nih.gov/pubmed/27054338 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2016.89 |
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