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Nicotinamide mononucleotide adenylyltransferase promotes hypoxic survival by activating the mitochondrial unfolded protein response

Gain-of-function mutations in the mouse nicotinamide mononucleotide adenylyltransferase type 1 (Nmnat1) produce two remarkable phenotypes: protection against traumatic axonal degeneration and reduced hypoxic brain injury. Despite intensive efforts, the mechanism of Nmnat1 cytoprotection remains elus...

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Bibliografske podrobnosti
izdano v:Cell Death Dis
Main Authors: Mao, X R, Kaufman, D M, Crowder, C M
Format: Artigo
Jezik:Inglês
Izdano: Nature Publishing Group 2016
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC4849163/
https://ncbi.nlm.nih.gov/pubmed/26913604
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2016.5
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