Hepatitis B virus genome replication triggers toll-like receptor 3-dependent interferon responses in the absence of hepatitis B surface antigen

The hepatitis B virus (HBV) has been described as stealth virus subverting immune responses initially upon infection. Impaired toll-like receptor signaling by the HBV surface antigen (HBsAg) attenuates immune responses to facilitate chronic infection. This implies that HBV replication may trigger ho...

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Bibliografski detalji
Izdano u:Sci Rep
Glavni autori: Real, Catherine Isabell, Lu, Mengji, Liu, Jia, Huang, Xuan, Trippler, Martin, Hossbach, Markus, Deckert, Jochen, Jahn-Hofmann, Kerstin, Ickenstein, Ludger Markus, John, Matthias Johannes, Gibbert, Kathrin, Dittmer, Ulf, Vornlocher, Hans-Peter, Schirmbeck, Reinhold, Gerken, Guido, Schlaak, Joerg Friedrich, Broering, Ruth
Format: Artigo
Jezik:Inglês
Izdano: Nature Publishing Group 2016
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Article
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC4848479/
https://ncbi.nlm.nih.gov/pubmed/27121087
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/srep24865
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