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Inflammatory and neuropathic cold allodynia are selectively mediated by the neurotrophic factor receptor GFRα3
Tissue injury prompts the release of a number of proalgesic molecules that induce acute and chronic pain by sensitizing pain-sensing neurons (nociceptors) to heat and mechanical stimuli. In contrast, many proalgesics have no effect on cold sensitivity or can inhibit cold-sensitive neurons and dimini...
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| 出版年: | Proc Natl Acad Sci U S A |
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| 主要な著者: | , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
National Academy of Sciences
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4843462/ https://ncbi.nlm.nih.gov/pubmed/27051069 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1603294113 |
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