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Calcium signaling, excitability and synaptic plasticity defects in mouse model of Alzheimer’s disease
Alzheimer’s disease (AD) and aging result in impaired ability to store memories, but the cellular mechanisms responsible for these defects are poorly understood. Presenilin 1 (PS1) mutations are responsible for many early-onset familial AD (FAD) cases. The phenomenon of hippocampal long-term potenti...
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| Pubblicato in: | J Alzheimers Dis |
|---|---|
| Autori principali: | , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2015
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4814213/ https://ncbi.nlm.nih.gov/pubmed/25589721 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3233/JAD-142427 |
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