Sustained Glucocorticoid Exposure Recruits Cortico-limbic CRH Signaling to Modulate Endocannabinoid Function

Sustained exposure to stress or corticosteroids is known to cause changes in brain endocannabinoid (eCB) signaling, such that tissue contents of the eCBs N-arachidonylethanolamine (AEA) are generally reduced while 2-arachidonoylglycerol (2-AG) levels increase. These changes in eCB signaling are to b...

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Publicado no:Psychoneuroendocrinology
Main Authors: Gray, J. Megan, Wilson, Christopher D., Lee, Tiffany T.Y., Pittman, Quentin J., Deussing, Jan M., Hillard, Cecilia J., McEwen, Bruce S., Schulkin, Jay, Karatsoreos, Ilia N., Patel, Sachin, Hill, Matthew N.
Formato: Artigo
Idioma:Inglês
Publicado em: 2016
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4788523/
https://ncbi.nlm.nih.gov/pubmed/26821211
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.psyneuen.2016.01.004
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