Ultraviolet-induced mutations in Cockayne syndrome cells are primarily caused by cyclobutane dimer photoproducts while repair of other photoproducts is normal.

Lignende værker

• The xeroderma pigmentosum group C gene leads to selective repair of cyclobutane pyrimidine dimers rather than 6-4 photoproducts
  af: Emmert, Steffen, et al.
  Udgivet: (2000)
• U-U and T-T cyclobutane dimers have different mutational properties.
  af: Gibbs, P E, et al.
  Udgivet: (1993)
• Repair by human cell extracts of single (6-4) and cyclobutane thymine-thymine photoproducts in DNA.
  af: Szymkowski, D E, et al.
  Udgivet: (1993)
• Cyclobutane Pyrimidine Dimer Density as a Predictive Biomarker of the Biological Effects of Ultraviolet Radiation in Normal Human Fibroblast
  af: Sproul, Christopher D., et al.
  Udgivet: (2013)
• RNA Polymerase II Transcription Suppresses Nucleosomal Modulation of UV-Induced (6-4) Photoproduct and Cyclobutane Pyrimidine Dimer Repair in Yeast
  af: Tijsterman, Marcel, et al.
  Udgivet: (1999)