The Missing Link in Epstein-Barr Virus Immune Evasion: the BDLF3 Gene Induces Ubiquitination and Downregulation of Major Histocompatibility Complex Class I (MHC-I) and MHC-II

Lignende værker

• The Epstein-Barr Virus G-Protein-Coupled Receptor Contributes to Immune Evasion by Targeting MHC Class I Molecules for Degradation
  af: Zuo, Jianmin, et al.
  Udgivet: (2009)
• Immune responses to Epstein–Barr virus: molecular interactions in the virus evasion of CD8(+) T cell immunity
  af: Rowe, Martin, et al.
  Udgivet: (2010)
• Distinct Roles of Extracellular Domains in the Epstein-Barr Virus-Encoded BILF1 Receptor for Signaling and Major Histocompatibility Complex Class I Downregulation
  af: Fares, Suzan, et al.
  Udgivet: (2019)
• Induction of the Lytic Cycle Sensitizes Epstein-Barr Virus-Infected B Cells to NK Cell Killing That Is Counteracted by Virus-Mediated NK Cell Evasion Mechanisms in the Late Lytic Cycle
  af: Williams, Luke R., et al.
  Udgivet: (2015)
• The Epstein-Barr Virus-Encoded BILF1 Protein Modulates Immune Recognition of Endogenously Processed Antigen by Targeting Major Histocompatibility Complex Class I Molecules Trafficking on both the Exocytic and Endocytic Pathways
  af: Zuo, Jianmin, et al.
  Udgivet: (2011)