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Autophosphorylation of αCaMKII downregulates excitability of CA1 pyramidal neurons following synaptic stimulation

It has been well documented that α-calcium/calmodulin-dependent protein kinase II (αCaMKII) is central to synaptic plasticity such as long-term potentiation, an activity-dependent strengthening of synapses that is thought to underlie certain types of learning and memory. However, the mechanisms by w...

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Bibliographische Detailangaben
Veröffentlicht in:Neurobiol Learn Mem
Hauptverfasser: Sametsky, Evgeny A., Disterhoft, John F., Ohno, Masuo
Format: Artigo
Sprache:Inglês
Veröffentlicht: 2009
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4692356/
https://ncbi.nlm.nih.gov/pubmed/19245842
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nlm.2009.02.006
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