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Autophosphorylation of αCaMKII downregulates excitability of CA1 pyramidal neurons following synaptic stimulation
It has been well documented that α-calcium/calmodulin-dependent protein kinase II (αCaMKII) is central to synaptic plasticity such as long-term potentiation, an activity-dependent strengthening of synapses that is thought to underlie certain types of learning and memory. However, the mechanisms by w...
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| Publicado no: | Neurobiol Learn Mem |
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| Main Authors: | , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2009
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4692356/ https://ncbi.nlm.nih.gov/pubmed/19245842 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nlm.2009.02.006 |
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