A single bacterial immune evasion strategy dismantles both MyD88 and TRIF signalling pathways downstream of TLR4

During bacterial infections, Toll-like receptor 4 (TLR4) signals through the MyD88-dependent pathway to promote rapid pro-inflammatory responses, but also signals via the TRIF-dependent pathway, which promotes Type-I interferon responses and acts with MyD88 signaling to potentiate inflammatory cytok...

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Detalhes bibliográficos
Publicado no:Cell Host Microbe
Main Authors: Rosadini, Charles V., Zanoni, Ivan, Odendall, Charlotte, Green, Erin R., Paczosa, Michelle K., Philip, Naomi H., Brodsky, Igor E., Mecsas, Joan, Kagan, Jonathan C.
Formato: Artigo
Idioma:Inglês
Publicado em: 2015
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4685476/
https://ncbi.nlm.nih.gov/pubmed/26651944
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.chom.2015.11.006
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