Altering central nervous system physiology with a defective herpes simplex virus vector expressing the glucose transporter gene.
Because of their postmitotic nature, neurons are difficult subjects for gene transfer. To circumvent this, we have used a defective herpes simplex virus vector to overexpress the rat brain glucose transporter (GT) gene under the control of the human cytomegalovirus ie1 promoter. This vector, designa...
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| Publicado no: | Proc Natl Acad Sci U S A |
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| Principais autores: | , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
1993
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC46360/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8386379/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.90.8.3655 |
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