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Antiphospholipid Antibodies Promote the Release of Neutrophil Extracellular Traps: A New Mechanism of Thrombosis in the Antiphospholipid Syndrome

OBJECTIVE: Antiphospholipid antibodies (aPL), especially those targeting beta-2-glycoprotein I (β(2)GPI), are well known to activate endothelial cells, monocytes, and platelets, with prothrombotic implications. In contrast, the interaction of aPL with neutrophils has not been extensively studied. Ne...

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محفوظ في:

التفاصيل البيبلوغرافية
الحاوية / القاعدة:	Arthritis Rheumatol
المؤلفون الرئيسيون:	Yalavarthi, Srilakshmi, Gould, Travis J., Rao, Ashish N., Mazza, Levi F., Morris, Alexandra E., Núñez-Álvarez, Carlos, Hernández-Ramírez, Diego, Bockenstedt, Paula L., Liaw, Patricia C., Cabral, Antonio R., Knight, Jason S.
التنسيق:	Artigo
اللغة:	Inglês
منشور في:	2015
الموضوعات:	Article
الوصول للمادة أونلاين:	https://ncbi.nlm.nih.gov/pmc/articles/PMC4626310/ https://ncbi.nlm.nih.gov/pubmed/26097119 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/art.39247
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Antiphospholipid Antibodies Promote the Release of Neutrophil Extracellular Traps: A New Mechanism of Thrombosis in the Antiphospholipid Syndrome

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