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Gilles de la Tourette syndrome is not linked to contactin-associated protein receptor 2 antibodies

BACKGROUND: In Gilles de la Tourette syndrome (GTS) an immunopathogenic influence of autoantibodies is suspected. In familial GTS a disruption of the contactin-associated protein 2 gene (CNTNAP2), coding for the contactin-associated protein 2 (CASPR2), has been reported. Autoantibodies against CASPR...

Täydet tiedot

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Bibliografiset tiedot
Julkaisussa:Mol Brain
Päätekijät: Sühs, Kurt-Wolfram, Skripuletz, Thomas, Pul, Refik, Alvermann, Sascha, Schwenkenbecher, Philipp, Stangel, Martin, Müller-Vahl, Kirsten
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: BioMed Central 2015
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4604618/
https://ncbi.nlm.nih.gov/pubmed/26462472
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13041-015-0154-6
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