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Synthetic dosage lethality in the human metabolic network is highly predictive of tumor growth and cancer patient survival

Synthetic dosage lethality (SDL) denotes a genetic interaction between two genes whereby the underexpression of gene A combined with the overexpression of gene B is lethal. SDLs offer a promising way to kill cancer cells by inhibiting the activity of SDL partners of activated oncogenes in tumors, wh...

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Bibliografiske detaljer
Udgivet i:Proc Natl Acad Sci U S A
Main Authors: Megchelenbrink, Wout, Katzir, Rotem, Lu, Xiaowen, Ruppin, Eytan, Notebaart, Richard A.
Format: Artigo
Sprog:Inglês
Udgivet: National Academy of Sciences 2015
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4593091/
https://ncbi.nlm.nih.gov/pubmed/26371301
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1508573112
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