Prenatal alcohol exposure alters methyl metabolism and programs serotonin transporter and glucocorticoid receptor expression in brain

Prenatal alcohol exposure (PAE) programs the fetal hypothalamic-pituitary-adrenal (HPA) axis, resulting in HPA dysregulation and hyperresponsiveness to stressors in adulthood. Molecular mechanisms mediating these alterations are not fully understood. Disturbances in one-carbon metabolism, a source o...

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Pubblicato in:Am J Physiol Regul Integr Comp Physiol
Autori principali: Ngai, Ying Fai, Sulistyoningrum, Dian C., O'Neill, Ryan, Innis, Sheila M., Weinberg, Joanne, Devlin, Angela M.
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Physiological Society 2015
Soggetti:
Hormones, Reproduction and Development
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4591382/
https://ncbi.nlm.nih.gov/pubmed/26180184
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpregu.00075.2015
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