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The key target of neuroprotection after the onset of ischemic stroke: secretory pathway Ca(2+)-ATPase 1
The regulatory mechanisms of cytoplasmic Ca(2+) after myocardial infarction-induced Ca(2+) overload involve secretory pathway Ca(2+)-ATPase 1 and the Golgi apparatus and are well understood. However, the effect of Golgi apparatus on Ca(2+) overload after cerebral ischemia and reperfusion remains unc...
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| Vydáno v: | Neural Regen Res |
|---|---|
| Hlavní autoři: | , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Medknow Publications & Media Pvt Ltd
2015
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4590240/ https://ncbi.nlm.nih.gov/pubmed/26487855 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4103/1673-5374.162760 |
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