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Repressed TGF-β signaling through CagA-Smad3 interaction as pathogenic mechanisms of Helicobacter pylori-associated gastritis
Helicobacter pylori (H. pylori) infection causes chronic gastric inflammation, peptic ulceration, and gastric carcinogenesis, in which H. pylori cytotoxin-associated gene A (CagA) plays major pathogenic action. Since transforming growth factor-β (TGF-β) and its signaling also are principally implica...
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| 出版年: | J Clin Biochem Nutr |
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| 主要な著者: | , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
the Society for Free Radical Research Japan
2015
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4566024/ https://ncbi.nlm.nih.gov/pubmed/26388668 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3164/jcbn.15-38 |
| タグ: |
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