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Repressed TGF-β signaling through CagA-Smad3 interaction as pathogenic mechanisms of Helicobacter pylori-associated gastritis
Helicobacter pylori (H. pylori) infection causes chronic gastric inflammation, peptic ulceration, and gastric carcinogenesis, in which H. pylori cytotoxin-associated gene A (CagA) plays major pathogenic action. Since transforming growth factor-β (TGF-β) and its signaling also are principally implica...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | J Clin Biochem Nutr |
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| Κύριοι συγγραφείς: | , , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
the Society for Free Radical Research Japan
2015
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4566024/ https://ncbi.nlm.nih.gov/pubmed/26388668 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3164/jcbn.15-38 |
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