Fatty acid binding protein deletion suppresses inflammatory pain through endocannabinoid/N-acylethanolamine-dependent mechanisms

BACKGROUND: Fatty acid binding proteins (FABPs) serve as intracellular carriers that deliver endocannabinoids and N-acylethanolamines to their catabolic enzymes. Inhibition of FABPs reduces endocannabinoid transport and catabolism in cells and FABP inhibitors produce antinociceptive and anti-inflamm...

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Dettagli Bibliografici
Pubblicato in:Mol Pain
Autori principali: Kaczocha, Martin, Glaser, Sherrye T., Maher, Thomas, Clavin, Brendan, Hamilton, John, O’Rourke, Joseph, Rebecchi, Mario, Puopolo, Michelino, Owada, Yuji, Thanos, Panayotis K.
Natura: Artigo
Lingua:Inglês
Pubblicazione: BioMed Central 2015
Soggetti:
Short Report
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4551694/
https://ncbi.nlm.nih.gov/pubmed/26311517
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12990-015-0056-8
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