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Diallyl disulfide inhibits TNFα induced CCL2 release through MAPK/ERK and NF-Kappa-B Signaling

TNFα receptors are constitutively overexpressed in tumor cells, correlating to sustain elevated NFκB and monocyte chemotactic protein-1 (MCP-1/CCL2) expression. The elevation of CCL2 evokes aggressive forms of malignant tumors marked by tumor associated macrophage (TAM) recruitment, cell proliferati...

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Detalhes bibliográficos
Publicado no:	Cytokine
Main Authors:	Bauer, D., Redmon, N., Mazzio, E., Taka, E., Reuben, JS., Day, A., Sadrud-Din, S., Flores-Rozas, H., Soliman, KFA., Darling-Reed, S.
Formato:	Artigo
Idioma:	Inglês
Publicado em:	2015
Assuntos:	Article
Acesso em linha:	https://ncbi.nlm.nih.gov/pmc/articles/PMC4532635/ https://ncbi.nlm.nih.gov/pubmed/26100848 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cyto.2014.12.007
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Diallyl disulfide inhibits TNFα induced CCL2 release through MAPK/ERK and NF-Kappa-B Signaling

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