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Astrocyte Calcium Signalling and Epilepsy

Studies performed over the last decade, in both animal models and human epilepsy, support the view that a defective K(+) buffering due to an altered expression of K(+) and aquaporin channels in astrocytes represents a possible causative factor of the pathological neuronal hyperexcitability in the ep...

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Publicado en:Glia
Autores principales: CARMIGNOTO, GIORGIO, HAYDON, PHILIP G.
Formato: Artigo
Lenguaje:Inglês
Publicado: 2012
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC4532388/
https://ncbi.nlm.nih.gov/pubmed/22389222
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/glia.22318
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