Pathogenesis of intracranial aneurysm is mediated by proinflammatory cytokine TNFA and IFNG and through stochastic regulation of IL10 and TGFB1 by comorbid factors

BACKGROUND: Intracranial aneurysm (IA) is often asymptomatic until the time of rupture resulting in subarachnoid hemorrhage (SAH).There is no precise biochemical or phenotype marker for diagnosis of aneurysm. Environmental risk factors that associate with IA can result in modifying the effect of inh...

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Veröffentlicht in:J Neuroinflammation
Hauptverfasser: Sathyan, Sanish, Koshy, Linda V., Srinivas, Lekshmi, Easwer, H. V., Premkumar, S., Nair, Suresh, Bhattacharya, R. N., Alapatt, Jacob P., Banerjee, Moinak
Format: Artigo
Sprache:Inglês
Veröffentlicht: BioMed Central 2015
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Research
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4510902/
https://ncbi.nlm.nih.gov/pubmed/26198819
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12974-015-0354-0
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