Absence of synaptotagmin disrupts excitation-secretion coupling during synaptic transmission.
Synaptotagmin is an integral synaptic vesicle protein proposed to be involved in Ca(2+)-dependent exocytosis during synaptic transmission. Null mutations in synaptotagmin have been made in Drosophila, and the protein's in vivo function has been assayed at the neuromuscular synapse. In the absence of...
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| Publicado no: | Proc Natl Acad Sci U S A |
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| Principais autores: | , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
1994
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC45095/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7938019/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.91.22.10727 |
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