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Mechanisms underlying presynaptic Ca(2+) transient and vesicular glutamate release at a CNS nerve terminal during in vitro ischaemia
KEY POINTS: Here we demonstrate presynaptic responses and mechanisms of increased vesicular glutamate release during in vitro ischaemia in the calyx of Held terminal, an experimentally accessible presynaptic terminal in the CNS. . The ischaemia-induced increase in presynaptic Ca(2+) was mediated by...
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| Izdano u: | J Physiol |
|---|---|
| Glavni autori: | , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
John Wiley & Sons, Ltd
2015
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4506182/ https://ncbi.nlm.nih.gov/pubmed/25833340 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/JP270060 |
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